Yang et al. [75] confirmed that hypoxia preconditioning of ASCs for 24 h led to the amplified release of HGF, IL-1, VEGF-A, FGF-2, and TGF-β, contributing to the prevention of apoptosis of cardiomyocytes through the JNK signaling pathway after MI injury. This evidence concerns the gene VEGFA and myocardial infarction.