Moreover, we found the same low levels of MGL mRNA in NAWM and rim sections of chronic inactive MS lesions, and although not significant, z-score values showed a clear difference in MGL expression, with higher expression of this lectin in the rim of active lesions compared to those from inactive lesions (Fig. 1a), suggesting a role for this endogenous lectin in areas of neuroinflammation. Here, CLEC10A is linked to myeloid sarcoma.