FH is caused by genetic mutations of the LDL-receptor (LDL-R), Apolipoprotein B (ApoB), proprotein convertase subtilisin/kexin type 9 (PCSK9) or low-density lipoprotein receptor adaptor protein 1 (LDLR-AP1) [4]. The gene discussed is APOB; the disease is familial hyperaldosteronism.