Moreover, we were able to demonstrate the extended molecular perspective of ATC with the support of our previously published RNA-seq data from 162 DTCs2; both BRAF-positive and RAS-positive ATCs presented a similar transcriptome profile, irrespective of their driver mutation, but were grouped into another subtype, ATC-like, rather than BRAFV600E-like. This evidence concerns the gene BRAF and Ehlers-Danlos syndrome, musculocontractural type.