ALT and AST levels could reinforce this hypothesis as they were found to be augmented in symptomatic vivax malaria patients when compared to HBV-monoinfected individuals (Table 2), while only being correlated to viremia in coinfected P. vivax-HBV patients and not in HBV-monoinfected subjects (S2 Fig). The gene discussed is GPT; the disease is Plasmodium vivax malaria.