Metabolic traits including fasting insulin (rG = 0.17, p = 0.53), homeostatic model assessment of insulin resistance (rG = 0.37, p = 0.48), fasting glucose (rG = 0.01, p = 0.97) and coronary artery disease (rG = −0.01, p = 0.97) were not genetically correlated with liver iron content. This evidence concerns the gene INS and coronary artery disorder.