Instead of the canonical neutrophilic response seen in wild-type infection, IL-33 treatment shifted the myeloid compartment toward dominant eosinophilia by day 2 post infection (57% with IL-33 vs. 20% without) and a trend toward increased numbers of eosinophils (Fig. 3b, c; Supplementary Fig. 6a, b). This evidence concerns the gene IL33 and Increased total eosinophil count.