MiR-34a, miR-182, and miR-144-3p levels are inversely correlated with MET levels in human gliomas and mechanistic studies have illustrated that they can specifically bind the MET 3′-untranslated region and inhibit its expression, thus potently repressing glioblastoma cell proliferation and invasion in vitro and in vivo [52–54]. Here, MET is linked to glioblastoma.