Other components of this model include the expression of molecules that are mechanistically linked to the priming of tumor-specific T cells by RT, such as cGAS and STING [55], and the expression of molecules that are linked to cancer-cell recognition by CD8 T cells, such as MHC-I, β2m, and components of the antigen-presentation machinery [92, 93]. This evidence concerns the gene STING1 and neoplasm.