Rosario et al. review the related papers and suggest that mutant FOXL2 maintains some of the transcriptional activity of the wildtype allele, but there is a subtle alteration of the expression in a unique suite of cancer-related genes, and they suggest that it is an oncogene or tumor suppressor, depending on the context related to the GCTs subtype [44]. Here, FOXL2 is linked to neoplasm.