JUN and breast carcinoma: As studies showed that DACH1 binds to c‐Jun and inhibits its function of contact‐independent growth in breast cancer cells,13, 26 and JNK/c‐Jun signaling pathway promoted cancer stem‐like cell (CSC) phenotype through Notch1 signaling in triple‐negative breast cancer (TNBC),27 we speculated that DACH1 might reverse EMT by suppressed Notch1 pathway via c‐Jun.