As studies showed that DACH1 binds to c‐Jun and inhibits its function of contact‐independent growth in breast cancer cells,13, 26 and JNK/c‐Jun signaling pathway promoted cancer stem‐like cell (CSC) phenotype through Notch1 signaling in triple‐negative breast cancer (TNBC),27 we speculated that DACH1 might reverse EMT by suppressed Notch1 pathway via c‐Jun. This evidence concerns the gene NOTCH1 and breast cancer.