We observed that stimulating Fcmr−/− BMDCs with 2′3′-cGAMP resulted in the upregulation of the T cell inhibitory receptor PD-L2 (Supplementary Fig. 5c), and that reduced tumor growth in Fcmr−/− DC transfer recipients was dependent on CD8+ T cells (Fig. 5h). This evidence concerns the gene CD8A and neoplasm.