VEGFA and neoplasm: Different biological mechanisms have been shown to explain thrombosis associated with cancer: expression of hemostatic proteins [74] or adhesion platelet molecules by tumor cells [75], production of procoagulant microparticles (MP) by tumor and/or host cells [76], inflammation [77], and the presence of other molecules such as some proangiogenic factors (VEGF, βTGF) [78].