Our observations stand in line with previous published results, showing a diminished IL‐29 response of neonatal mDCs in‐vitro after infection with human cytomegalovirus.22 These lower levels of IFNs cannot be explained by different cell counts of pDCs or BDCA3+ mDCs as shown in our previous study.4 Thus, this impaired IL‐29 secretion might be an intrinsic functional deficiency of the neonatal innate immunity. Here, THBD is linked to infection.