For instance, in breast cancer and prostate cancer, the activation of AKT1 can stimulate the proliferation of tumor cells, but at the same time it can suppress the migration ability of these cells (Gao, Alwhaibi, Artham, Verma, & Somanath, 2018; Riggio et al., 2017). This evidence concerns the gene AKT1 and prostate carcinoma.