Studies have found that abnormally elevated levels of multiple factors, including long noncoding RNAs, microRNAs, and transforming growth factor (TGF)-β, as well as signalling pathways such as the nuclear factor-κB, WNT, and NOTCH pathways, can activate EMT and promote metastasis in cervical cancer [5–10]. This evidence concerns the gene TGFB1 and cervical carcinoma.