A schematic diagram depicted Gal‐3 contribution to ovarian cancer progression (Figure 9) which showed that Gal‐3 could establish a reinforcing STAT3‐HIF‐1α positive feedback loop through integrins and/or cytokines which end‐up with increased levels of Gal‐3 leading to increased cell survival, proliferation, chemoresistance, migration, and invasion of ovarian cancer. This evidence concerns the gene LGALS3 and ovarian carcinoma.