Our results do not contradict the data of other researchers who consider that level of MCP-1 in atherosclerotic plaques, including those with calcinosis, is associated with instability of the plaque and that increased levels of MCP-1 in blood is key for identification of unstable plaques presence in patients with atherosclerosis with a high probability and connected with myocardial infarction development [13, 17, 18]. This evidence concerns the gene CCL2 and calcinosis.