Moreover, the applied therapy led to a different nature of changes in plasma concentrations of adipocytokines with anti-inflammatory activity, i.e., an increase in omentin-1 concentration and a decrease in CTRP3 concentration, reflecting the complex nature of the endocrine activity of fat tissue in regulating the inflammatory process associated with type 2 diabetes. The gene discussed is C1QTNF3; the disease is type 2 diabetes mellitus.