Our preliminary results of SLC26A4 testing suggest that HL of at least 4 out of 18 (22%) patients with monoallelic GJB2 mutations was caused by homozygosity or compound heterozygosity for SLC26A4 pathogenic variants (Table S1), and consequently these patients are definitely the coincidental carriers of one pathogenic GJB2 allele. This evidence concerns the gene SLC26A4 and Hodgkins lymphoma.