Through the analysis of the cellular immune response of the Sv129 and BALB/c mice, although the infection increased the numbers of IFN-γ-producing cells, we could conclude that in vivo infection did not prime the proliferation of IFN-γ-producing CD4+ and CD8+ T cells sufficient enough to contain the infection (Figures 3, 4). The gene discussed is IFNG; the disease is infection.