Additionally, through the production of the keratinocyte growth factor (KGF), vascular endothelial growth factor (VEGF), and hepatocyte growth factor (HGF), MSCs promoted the regeneration of ATII cells, prevented the apoptosis of endothelial cells, and contributed to the enhanced repair of the alveolar-epithelial barrier in the ARDS-injured lungs [32–34]. The gene discussed is FGF7; the disease is acute respiratory distress syndrome.