Peripheral blood sulfatide-reactive type II NKT cells in AIH patients produced mainly TNFα (48.5% of type II NKT cells were TNFα+) and, to a lesser extent, IFNγ (8.6% of type II NKT cells were IFNγ+), IL-17 (0.6% of type II NKT cells were IL-17+) or IL-4 (3% of type II NKT cells were IL-4+; Supplementary Figures S2A–D). The gene discussed is IL17A; the disease is autoimmune hepatitis.