This resulted in an increased NK cell activation (CD69, NKp30, NKp44, NKG2D) as well as an increased tumor-directed cytotoxicity against chronic myeloid leukemia (CML) blasts and the leukemia cell line K562 compared to those NK cells stimulated without slanMo (63, 65). This evidence concerns the gene NCR3 and chronic myelogenous leukemia, BCR-ABL1 positive.