We found that MS2-tagged wild-type SNHG1 (SNHG1-WT-MS2) was significantly enriched for miR-194 in glioma cells compared to the empty vector and SNHG1 with a mutation in the miR-194 binding site (SNHG1-MUT-MS2) (Fig. 5l). Here, ADAM8 is linked to glioma.