Giventhat lncRNAs MIAT and MALAT1 are known to affectendothelial function through alteration of specific signaling pathways that affectcellular proliferation, migration, and survival (Liuet al., 2014; Yanet al., 2015), we speculated on their potential rolein the development of atherosclerosis and subsequent CAD. The gene discussed is MIAT; the disease is coronary artery disorder.