MIAT and myocardial infarction: This may suggest that MIAT levels are not initially raisedduring the acute attack, but rise later and remain elevated for an extended time.The significance of this, however, is yet unclear, but may point to a possible roleof MIAT as an adaptive mechanism similar to the activation of therenin-angiotensin-aldosterone system that occurs in post MI patients (Sutton and Sharpe, 2000).