Giventhat lncRNAs MIAT and MALAT1 are known to affectendothelial function through alteration of specific signaling pathways that affectcellular proliferation, migration, and survival (Liuet al., 2014; Yanet al., 2015), we speculated on their potential rolein the development of atherosclerosis and subsequent CAD. This evidence concerns the gene MIAT and atherosclerosis.