Such epilepsies are essentially variants of autoimmune encephalitis, including anti-N-methyl-D-aspartate (NMDA)-receptor encephalitis or anti-leucine-rich glioma-inactivated-1 (LGI1) encephalitis, in which autoantibodies for autoantigens elicit excessive inflammation in the brain. This evidence concerns the gene LGI1 and viral encephalitis.