The pool of targets associated with resistance to DEHP in FVB/N were previously involved in sperm resistance mechanisms (Table 1), against bacterial infections or innate host defense (Defb30, Spag11b), in protective mechanisms against oxidative stress (Gpx5), and in the clearance of exogenous compounds (Lcn5, Lcn9, Lcn5) [44, 115] in accordance with previously reports [116, 117]. Here, LCN9 is linked to bacterial infectious disease.