In the case of TA, authors have demonstrated the proliferation of TCD3 lymphocytes against heat shock proteins (HSP) of mycobacterium HSP65 and its human homologue HSP60, and the presence of antibodies IgG against both proteins, suggesting previous infections, possibly as a result of shared epitopes of HSPs leading to autoimmunity [23]. The gene discussed is HSPD1; the disease is infection.