While, however, the rate of adipocyte apoptosis, based on the mRNA expression levels of Bid, was not higher in LXR agonist-treated wild type mice, their hepatic steatosis was significantly more pronounced than that of the LXR agonist-treated mice in which TG2 was absent from the bone marrow-derived cells. This evidence concerns the gene TGM2 and Hepatic steatosis.