Finally, despite the finding that CaMKK2 depletion has a positive impact on BMDM (e.g., enhanced T cell attracting ability and reduced capability to inhibit T cell proliferation), other myeloid cells within TME also express CaMKK2 and thus may also contribute to the tumor growth inhibition observed in CaMKK2-ablated hosts. This evidence concerns the gene CAMKK2 and neoplasm.