KRAS and gastric neoplasm: Amplifications mainly targeted well-known oncogenes such as EGFR, ERBB2, KRAS, MYC, CCNE1, JAK2, FGFR2 et al. 12 gastric tumor tissues harbored CCNE1 amplifications (7.84%), which was the most frequent genes with SCNA in this study, and with similar frequency (10.58%) in TCGA cohort [4].