The FLT3 receptor has been reported to be mutated in about 30% of AML patients [11], and two kinds of mutations may be identified: in-frame duplications within the juxtamembrane region (FLT3-ITD), which can be observed in about 25% of AML, and point mutations in the tyrosine kinase domain (FLT3-TKD), detected in 7% of the cases. Here, FLT3 is linked to acute myeloid leukemia.