They are (1) TTP, which is aberrant thrombosis because vessel is not injured, but still utilizes “ULVWF” path in ADAMTS13 deficiency, (2) APL, which is aberrant thrombosis because vessel is not injured, but still utilizes “TF” path due to overexpressed TF from leukemic promyelocytes, and (3) heparin-induced thrombocytopenia with thrombosis syndrome, which is pathologic thrombosis because vessel is not injured, and also utilizes neither ULVWF path nor TF path [4, 20]. This evidence concerns the gene ADAMTS13 and acute promyelocytic leukemia.