We characterized the extent and uniformity of transcriptional effects of BETi in medulloblastoma models through expression profiling of four MYC-driven medulloblastoma cell lines treated with the BET-bromodomain inhibitor JQ1, relative to vehicle controls (Supplementary Data File 1 and Supplementary Figs. 1 and 2). This evidence concerns the gene MYC and medulloblastoma.