TGFB1 and uterine corpus leiomyoma: Patterns of inverse association of miRs with mRNA expression in uterine leiomyomas revealed an involvement of multiple candidate pathways, including extensive transcriptional reprogramming, cell proliferation control, decreased programmed cell death, mitogen-activated protein kinase (MAPK), TGF-β, WNT, Janus kinase/signal transducers and activators of transcription signaling, remodeling of cell adhesion, and cell–cell and cell–matrix interactions [19].