KCNA3 and stroke disorder: Based on our flow cytometric and immunohistochemical results as well as the recently published efficacy of selective Kv1.3 channel blockers in the MCAO mouse model [14], the pattern of functional Kv1.3 channel expression following tMCAO indicates that microglia that highly express Kv1.3 channels at 24–72 h post-tMCAO, but not infiltrating monocytes/macrophages, are likely to be the targets of Kv1.3-blocking therapies in stroke.