We also found that BS008 exerts an effect on AS of these gene transcripts in K562 leukemia cells by detecting an increase in the proapoptotic splice variants BCL‐XS, SMAC‐3, and HIPK3 U(−) with a concomitant decrease in the antiapoptotic splice variant HIPK3 U(+). Here, BCL2L1 is linked to leukemia.