First, we only controlled for the presence of the Hb EE homozygous genotype in women in our trial, as it has shown to be the strongest negative predictor of maternal Hb in the study population (Karakochuk et al., 2015); however, we acknowledge that there are other known genetic Hb disorders that may have affected biochemical results (e.g., Hb E heterozygous or α‐thalassemia). This evidence concerns the gene GSTM1 and alpha thalassemia spectrum.