Next, we hypothesized that CLEC‐1B, a C‐type lectin‐like receptor that is expressed on NK cells, could be a HRG receptor on NK cells based on previous reports suggesting a relationship between CLEC‐1B and tumor immunity.35 Due to an abundance of potential epitopes, we used human anti‐CLEC polyclonal antibodies to examine the effect of HRG on NK cell PD‐1 expression. The gene discussed is CLEC1B; the disease is neoplasm.