Bortezomib was reported that it could attenuate renal fibrosis in mice via the suppression of TGF‐β1 in UUO model.23 Our vivo study in rats showed that Bortezomib could not effectively reduce the expression and secretion of TGF‐β1in allograft kidney (Figure S3C‐E). The gene discussed is TGFB1; the disease is renal fibrosis.