TSLPR−/− mice have been shown to produce more IFN-γ in an experimental modal of Trypanosoma congolense infection (45), more IFN-γ and IL-17 in an inducible modal of colitis (46) compared to TSLPR+/+ mice and IL-1β is crucial for the generation of inflammatory IFN-γ/IL-17 double producing T cells during C. albicans infection (31, 42). The gene discussed is IL1B; the disease is colitis.