Since most of their ASD-like phenotypes are sexually dimorphic, Crmp4-KO mice may represent a powerful model for investigating the pathogenesis of ASD and the prenatal sex steroid theory of autism in addition to Crmp4-KO mice possibly providing an animal model for investigating some other developmental disorders including ADHD and learning disabilities associated with sensory processing issues. Here, DPYSL3 is linked to attention deficit-hyperactivity disorder.