Most notably, reeler recapitulates phenotypes observed in GM130 KO mice (Liu et al., 2017) including ataxia and cerebellar atrophy (Yuasa et al., 1993; Magdaleno et al., 2002) suggesting that Reelin participates in pathways associated with Golgi structural regulation during early brain development. The gene discussed is GOLGA2; the disease is cerebellar ataxia.