Accordingly, covariance structure analyses using path models A or C revealed that high BNP was a significant cause of the more pronounced decreases that were observed in the glucose and HOMA-IR levels of patients with acute myocardial infarction (path from BNP to glucose; β = −0.224, P = 0.004 and to HOMA-IR; β = −0.266, P = 0.012), whereas the finding that high noradrenaline was a significant cause of the consistent increases in both parameters (path from noradrenaline to glucose; β = 0.306, P < 0.001 and to HOMA-IR; β = 0.217, P = 0.04). Here, NPPB is linked to acute myocardial infarction.