PRRT2 was recently identified as a causative gene of paroxysmal kinesigenic dyskinesia (PKD) and other paroxysmal disorders, including benign familial infantile seizures (BFIS), infantile convulsions with choreoathetosis (ICCA), and paroxysmal hypnogenic dyskinesia (PHD).1, 2, 3, 4 Among these PRRT2‐related disorders, PKD is the most common phenotype, which is a dominantly hereditary disorder characterized by short and recurrent attacks triggered by a sudden initiation or alteration of voluntary movement.5 Age at onset (AAO) is usually during childhood or early adulthood. This evidence concerns the gene PRRT2 and episodic kinesigenic dyskinesia 1.