Interestingly, such complex overlapping features including FA and other DNA‐repair disorders were described in a recent study that reported a malfunction of the nuclease ERCC1‐ERCC4 (XPF) resulting in complex clinical manifestations comprising Xeroderma pigmentosum (XP), Cockayne syndrome (CS), and Fanconi anemia (FA) (Kashiyama et al., 2013). This evidence concerns the gene ERCC1 and Friedreich ataxia.