c‐Myc has been shown to be a protooncogene that modulates numerous cancer cell behaviors, including proliferation, differentiation, migration, and genomic instability.33, 34, 35 Recently, Ma et al demonstrated that c‐Myc also suppressed cell migration in Drosophila and human lung adenocarcinoma cell lines in a JNK‐dependent manner.36 Consistent with the results of that research, our results indicated that suppressed JNK activity was positively correlated with c‐Myc expression. The gene discussed is MAPK8; the disease is lung adenocarcinoma.