To examine whether IL-1β-induced iNOS expression and NO production are involved in proparasitic functions in A172 glioblastoma, IMR-32 neuroblastoma, and T98G glioblastoma cells, we compared the IFN-γ-mediated reduction of T. gondii numbers in the presence or absence of IL-1β (Figure 2A). Here, IL1B is linked to neuroblastoma.