The LN-resident DC subsets in question are CD1a−CD11c+CD14− conventional LN-resident DC (LNRcDC) and the CD123+BDCA-2 plasmacytoid DC (pDC) subset which have been shown to be involved in the cross-priming and type-I IFN-driven boosting of cytotoxic T cell responses against tumor-derived antigens, respectively [34, 35]. This evidence concerns the gene CD14 and neoplasm.